Peptides: CJC-1295 Overview (DAC vs No-DAC)

Category: growth-hormone Updated: 2026-04-04

CJC-1295 with DAC produced sustained IGF-1 elevation over 14 days in healthy adults (PMID 16368745). The no-DAC form preserves pulsatile GH secretion, preferred for most protocols.

Key Data Points
MeasureValueUnitNotes
Evidence GradeBgradeHuman pharmacokinetic data for both forms (PMID 16968793; 16368745); no body composition RCTs at performance protocol doses
CJC-1295 with DAC — Half-life6–8daysDrug Affinity Complex (DAC) modification enables albumin binding; dramatically extends circulating half-life
CJC-1295 without DAC — Half-life~30minutes (subcut)Also called Mod GRF 1-29; 29-amino acid GHRH analogue; short half-life preserves pulsatile GH pattern
IGF-1 Elevation (DAC form)1.5–3×above baselineSustained IGF-1 elevation maintained for 14+ days with weekly DAC injections (PMID 16368745); blunts pulsatility
Standard No-DAC Dose100–200mcg/injectionInjected simultaneously with GHRP; 2–3× daily; timing must match GHRP injection exactly
Standard DAC Dose1–2mg/injectionOnce or twice weekly; much larger per-dose due to sustained-release kinetics; NOT used in typical pulsatile protocols
Synergy with GHRPs3–4×greater GH pulseCJC-1295 no-DAC + ipamorelin or GHRP-2 injected simultaneously produces approximately 3–4× greater GH pulse than either alone

CJC-1295 is a synthetic GHRH analogue, but the term is often used loosely to refer to two fundamentally different compounds: the DAC form (with the Drug Affinity Complex modification) and the no-DAC form (also called Mod GRF 1-29). This distinction is not cosmetic — it determines the pharmacokinetics, GH secretion pattern, dosing protocol, and appropriateness for different use cases. Confusing the two forms is one of the most common errors in GH peptide protocols.

The Two Forms: A Critical Distinction

Native GHRH (Growth Hormone Releasing Hormone, 44 amino acids) has a plasma half-life of only 7 minutes due to enzymatic cleavage by dipeptidyl peptidase IV (DPP-IV). CJC-1295 was designed to solve this by substituting amino acids at key cleavage sites, producing a 29-amino acid GHRH fragment (the “Mod GRF 1-29” core) with approximately 30-minute half-life after subcutaneous injection.

The DAC modification adds a reactive maleimidyl group that covalently binds to albumin’s free cysteine residue (Cys34), creating a circulating reservoir with a 6–8 day half-life.

Form Comparison Table

FormHalf-lifeGH Pattern ProducedTypical DoseInjection FrequencyPreferred For
CJC-1295 with DAC6–8 daysSustained, non-pulsatile elevation (blunted pulsatility)1–2mgOnce or twice weeklyMaximizing IGF-1; less concern for pulsatility
CJC-1295 without DAC (Mod GRF 1-29)~30 min (subcut)Clean pulsatile GH pulse when combined with GHRP100–200mcg2–3× daily, simultaneous with GHRPMost performance and optimization protocols
Native GHRH 1-44 (reference)~7 minVery brief GH pulse; not practical for protocolsN/ANot typically usedResearch reference only
Sermorelin (GHRH 1-29)~10–12 min (subcut)Pulsatile; shorter duration than no-DAC CJC-1295100–200mcg2–3× dailyClinical GH deficiency (FDA-approved at one time)

Human Evidence

Teichman et al. 2006 (PMID 16368745): CJC-1295 with DAC administered to healthy adults produced dose-dependent IGF-1 elevation of 1.5–3× above baseline, maintained for 14+ days after a single injection. This is the primary human pharmacokinetic study for the DAC form.

Ionescu & Frohman 2006 (PMID 16968793): Demonstrated that pulsatile GH secretion persists during continuous GHRH receptor stimulation — supporting the mechanistic rationale for pulsatile protocols using the no-DAC form.

Why Pulsatile GH Matters

Continuous GH elevation (as with the DAC form) shifts GH signaling away from pulsatile patterns associated with anabolic and lipolytic effects. Pulsatile GH preferentially:

  • Drives hepatic IGF-1 production in discrete pulses
  • Promotes lipolysis in adipose tissue
  • Minimizes insulin resistance compared to continuous GH elevation
  • Reduces risk of GH receptor desensitization over time

This is why the no-DAC form (Mod GRF 1-29) is the standard in performance protocols: it amplifies the body’s natural pulsatile GH rhythm rather than replacing it with continuous elevation.

JurisdictionStatusScheduleNotes
USAResearch chemicalUnscheduledNot FDA-approved; WADA prohibited (S2) for competitive athletes
UKNot scheduledNoneLegal to possess; WADA prohibited under UKAD anti-doping rules
AustraliaPrescription restrictedSchedule 4 (TGA)ASADA prohibited; not commercially available without prescription
CanadaGray marketNo scheduleNo approved DIN; WADA prohibited for athletes
EUGenerally unscheduledVaries by countryNo EMA approval; WADA prohibited for athletes
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Frequently Asked Questions

What is the critical difference between CJC-1295 with DAC and without DAC?

The DAC (Drug Affinity Complex) modification causes CJC-1295 to bind covalently to circulating albumin, extending its half-life from ~30 minutes to 6–8 days. This creates sustained, non-pulsatile GH stimulation. The no-DAC form (Mod GRF 1-29) has a ~30-minute half-life and produces a clean GH pulse when injected. Most performance protocols use the no-DAC form specifically because pulsatile GH is more physiological and associated with better body composition outcomes.

Can CJC-1295 with DAC and GHRPs be combined?

Yes, but the combination produces a different GH pattern. CJC-1295 with DAC provides ongoing GHRH-receptor stimulation, while GHRPs continue to trigger pulsatile releases on top of the sustained baseline. Some practitioners use this approach for IGF-1 maximization, but it is less physiological than the no-DAC + GHRP combination. The Ionescu & Frohman 2006 study (PMID 16968793) confirmed that pulsatile GH secretion persists even during continuous GHRH stimulation.

Why is CJC-1295 without DAC (Mod GRF 1-29) preferred in most protocols?

Pulsatile GH secretion is the physiological norm, and pulsatile GH patterns are associated with superior anabolic signaling compared to continuous elevation. Injecting CJC-1295 no-DAC simultaneously with a GHRP produces a discrete, large GH pulse — mimicking (and amplifying) the body's natural pulsatile rhythm. The short 30-minute half-life means the system clears quickly, allowing normal GH regulation between doses and reducing the risk of receptor desensitization.

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